Zentel 400mg tablet-1’s

Zentel 400mg is a broad-spectrum anthelmintic used to treat various parasitic worm infections, including roundworms, hookworms, tapeworms, and more. It is especially effective in areas with high worm infestation rates, such as Africa and Tanzania, improving overall health by eliminating parasites and reducing the risk of complications like malnutrition or intestinal blockages.

Available Product Formulations

• Zentel 400mg Tablet (single-dose tablet).
• Zentel Suspension (liquid formulation for children or individuals with swallowing difficulties).

Uses

• Treatment of intestinal parasitic infections such as ascariasis, trichuriasis, and hookworm infections.
• Management of systemic parasitic infections, including hydatid disease and neurocysticercosis.
• Preventive therapy in endemic regions with recurrent worm infestations.

How to Use

• Dosage: Adults and children over 2 years: 1 tablet (400mg) as a single dose.
• Duration: Typically one-time use; repeated only if recommended by a healthcare provider.

Side Effects

• Common: Nausea, abdominal pain, dizziness, and headache.
• Rare: Allergic reactions like rash, itching, or liver enzyme changes.
  Seek medical advice if severe or persistent side effects occur.

Interactions

• May interact with medications like cimetidine, dexamethasone, or antiepileptic drugs, potentially altering its effectiveness.
• Best taken with food to enhance absorption and efficacy.
• Discuss with a healthcare provider if combining with other medications or supplements.

Warnings

• Avoid during pregnancy or in women planning to conceive due to potential teratogenic effects.
• Use cautiously in individuals with liver disease or pre-existing health conditions.
• Not suitable for children under 2 years unless directed by a healthcare provider.
• Maintain proper hygiene to prevent reinfection.

Table of Important Information

How Zentel 400mg Tablet Works

Zentel’s active ingredient, Albendazole, is a broad-spectrum anthelmintic. It disrupts the parasite’s cytoskeleton by inhibiting tubulin polymerization, leading to the loss of cytoplasmic microtubules. This action impairs the parasite’s ability to absorb nutrients, causing its death and subsequent elimination from the body.